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LGD-4033 boasts high selectivity when it bonds to androgen-receptive cells in the body, opting for those in muscles and bonesbut not in fat, but the enzyme has never been used to treat obesity, let alone obesity and cancer."This could really open up a new approach to treating cancer, because, in some cancers, the enzyme turns down, but in others it has to be turned back on," explains David Schutte, one-time head of the National Cancer Institute, and co-senior author of the paper.For several years, Schutte and his team have been trying to see how long such a molecule can stay in one area, even if other areas of the body switch off as a result.Schutte and his team made the finding after studying mice that developed melanoma, another type of cancer, by genetically modifying them. Mice lacking melanoma had an enzyme gene in their bone marrow to turn off the enzyme, making it unable to attach to androgen-receptors and thus unable to fight the bacteria that cause melanoma.After about several years of observation, they discovered that the mice could eventually return to normal if the enzyme was reintroduced."It is not yet known if these mice can maintain normal levels of enzyme activity in other locations and whether this could be useful in humans, but this work is certainly a first step," says co-lead author Paul Whelan from the University of Aberdeen, UK."The enzyme is not yet understood well enough to be used to combat any of the diseases of concern that could result from its overexpression, such as obesity, but it certainly opens up a lot of possibilities to investigate further."Similar articles: